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Coverage for GLP-1 medications for obesity is expanding, but remains patchy

ATP channel, increasing K ATP channel sensitivity to ATP and leading to K ATP channel closure, cell membrane depolarization and VDC channel opening, followed by Ca 2+ influx through VDC channels into cells to promote the exocytosis of insulin granules and the acute secretion of insulin and into circulation (Mller et al., 2019) (Figure 2)

This is particularly relevant when considered alongside GLP-1 receptor agonists, which target the downstream metabolic consequences of estrogen withdrawal directly

ILC2s respond rapidly to epithelial-derived alarmins such as IL-33, IL-25, and thymic stromal lymphopoietin, producing large quantities of IL-5 and IL-13 independently of adaptive immune system (5, 6)