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The First Generation: GLP-1 Mono-Agonists The first major breakthrough was Exenatide (Byetta), followed by Liraglutide (Saxenda)
Insulin and IGF-1 improve mitochondrial function in a PI-3K/Akt-dependent manner and reduce mitochondrial generation of reactive oxygen species in Huntingtons disease knock-in striatal cells
Sterile water (no preservative) drops this to ~24 hours
These medications work by mimicking the actions of the GLP-1 hormone, which is naturally produced in the body and plays several key roles in appetite regulation and glucose metabolism
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