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Across analyses, a recurring contrast is observed between (i) a reproductive subtypeoften reported as higher LH and SHBG with elevated androgens despite comparatively lower BMI and fasting insulinand (ii) a metabolic subtype, characterized by higher adiposity with higher glucose/insulin measures and lower SHBG (and, in some reports, lower LH) (51)
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Conversely, an overabundance of saturated fat has been associated with amplified androgen levels and metabolic disorders in PCOS [20]
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