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Saunte et al., 2017), suggesting that the induced overactive Th17 response is a potential pathogenic factor of psoriasis

More long-term studies in humans are needed

Notably, we have revealed here that sustained intracellular GSH levels are essential for CD8 + T cell expansion, survival, and effector function and defined the complete dependence of such regulation on GPX4 expression and activity, through a metabolic signaling axis that ties the A2AR/PKA/E2F1 pathway to GSH metabolism and lipid peroxidation