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Mice deficient in endothelial NO synthase (eNOS), the key enzyme for NO production in vessels, are characterized by faster erythrocyte clearance, possibly due to enhanced erythrocyte PS exposure and subsequent phagocytosis [170]

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In people without pernicious anemia, about 0.5 g/day of B12 is reabsorbed by enterohepatic circulation in which B12-containing bile is secreted by the liver into the digestive tract where the B12 is then reabsorbed via intrinsic factor

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