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GPX4 inhibition with RSL3 induces ferroptosis, lipid peroxidation and reactive oxygen species (ROS) in thyroid cancer cells in vitro We targeted the ferroptosis pathway in PTC cells in vitro using a pharmacologic inhibitor of GPX4, RSL3
N237S) disorder characterized by an increase in reactive oxygen species (ROS), chronic loss of axons and progressive loss of nervous system function in pediatric patients
High serum homocysteine levels may also be important in the pathogenesis of colon cancer, diabetic retinopathy, and other diseases
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