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The question: does sustained elevation of intracellular NAD+ via NNMT inhibition produce the same long-term benefits as NAD+ precursor supplementation (like nicotinamide riboside or NMN), or are there trade-offs we haven't identified yet
A Synergistic Approach: AHK-Cu is a powerful tool, but it doesn't have to work alone
Maintain fasted administration
A lack of this important nutrient can cause tiredness, brain changes, and big red blood cells
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