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High-salt diet does not boost neuroinflammation and neurodegeneration in a model of -synucleinopathy
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Metaphases analysis To assess single-chromosome damage, HCT116 Wild type (WT) and ADH5 cells were plated in P60 dishes, allowed to adhere and then treated with mitomycin C (MMC, Santa Cruz, #sc-3514) 0.5 g mL 1 during 24 h or with FA 150 mol L 1 during 48 h
79 Rather than depending on a single relay mechanism, extracellular ferroptotic propagation is likely mediated by a context-dependent signaling network whose dominant components vary with tissue architecture and disease state
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