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Site-direct mutagenesis Site-direct mutagenesis was used to generate the proIGF-1Ea N92D mutant lacking the glycosylation site

The formulation was designed for researchers studying multi-pathway approaches to tissue regeneration and skin repair, where the mechanisms of all three compounds may interact

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A 2000 study on female rats found that 12 weeks of this compound increased bone mineral content in the tibia and vertebrae, though this was linked to overall body weight gain, indicating its effects may be tied to general growth promotion (10)