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Mechanism of Action How Dihexa activates the HGF/c-Met pathway to promote synaptogenesis, dendritic spine formation, and long-term potentiation

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Cellular-Response Research Research involving GHK-Cu has explored its interaction with: Cellular-repair and regenerative signaling pathways Oxidative-stress and antioxidant-defense mechanisms Tissue-remodeling and extracellular-matrix pathways Cellular-response and physiological-maintenance signaling Because GHK-Cu interacts with regenerative and growth-related pathways, some research settings involving active cancer-, tumor-, or abnormal proliferative research models may warrant additional monitoring and individualized evaluation

8 , e1000368 (2010)