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Recently, a groundbreaking therapeutic strategy utilizing molybdenum disulfide (MoS2) as a carrier for the delivery of anti-PD-L1 antibody and V9302, a competitive transmembrane antagonist targeting the amino acid transporter ASCT2, has demonstrated remarkable potential in enhancing anti-tumor immune responses within TNBC cells [194]

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This highlights the indispensable role of cAMP signaling in the efficacy of GLP1R agonism and underscores that targeting this secondary messenger system could be critical for optimizing therapeutic interventions