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I mean of course that makes sense of the pathways, but I did not
Similar content being viewed by others Subjects Introduction Cardiovascular complications from cancer therapies are highly prevalent, making cardiotoxicity the second leading cause of morbidity and mortality among cancer survivors 1,2
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In PCa cultured cells ER/eNOS causes GSTP1 repression by being recruited at estrogen responsive elements within its promoter favoring a local chromatin remodeling together with hypermethylated promoter sequences
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