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In this issue, the already mentioned Tkalcevic and collaborators study may be illustrative (Seveljevic-Jaran et al., 2006

The addition of glucagon receptor agonism introduces theoretical concerns about hepatic glucose output, though no clinically significant hyperglycemia was observed

In another, a lifespan trial in Swiss-derived SHR mice (1 g/mouse per month) reported no changes in food intake or body weight, while chromosomal aberrations decreased and maximum lifespan increased [7]

1 Furthermore, the advent of PCSK9-inhibiting drugs in the mid-2010s paved the way for further LDL lowering and risk reduction in people whose cholesterol is not adequately controlled by statins, although cost remains a barrier