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Consequently, many GSH-responsive nanoplatforms are designed either to deplete GSH by targeting these pathways or to exploit high GSH levels as triggers for selective drug release
Role of Endoplasmic Reticulum Stress Induction by the Plant Toxin, Persin, in Overcoming Resistance to the Apoptotic Effects of Tamoxifen in Human Breast Cancer Cells
Briefly, equal amount of samples were loaded onto 12% SDSPAGE gels, transferred to polyvinylidene difluoride (PVDF) membranes and analyzed using antibody against GPX4 at a concentration of 1:2,000
Toxicology
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