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EhlersDanlos syndrome due to tenascin-X deficiency: muscle weakness and contractures support overlap with collagen VI myopathies
Infiltrating M1 macrophages polarized via either Notch/NF-B crosstalk or prostaglandin receptor EP3/Rho kinase signaling secrete pro-inflammatory cytokines that exacerbate tubular necroptosis, extracellular matrix deposition, and renal fibrosis (136, 137)
Its small size and copper-binding affinity underpin its bioactivity, distinguishing it from larger proteins or synthetic peptides
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