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6-phosphogluconolactone is then converted to 6-phosphogluconate via the lactonase enzyme
These findings provide a molecular basis for the elucidation of the effects caused by riboflavin deficiency
These changes collectively lead to adipose tissue dysfunction, characterized by increased release of inflammatory cytokines, enhanced lipolysis, impaired insulin signaling, and the development of leptin resistance, ultimately leading to the typical metabolic abnormal phenotype characteristic of PCOS (51, 5355)
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