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Toxic Mechanism: The main toxicity following paracetamol poisoning is acute liver injury which results from the formation of a toxic metabolite of paracetamol, N-acetyl-p-benzoquinone imine (NAPQI)

Over the long term, elevated cortisol from chronic physical or psychological stress compromises health, in part by promoting insulin resistance and accumulation of visceral fat (Lehrke 2008

This involves a rigorous review of studies, including those with potential biases favouring the arginine variant and weighing them against independent findings

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