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This disrupts calcium homeostasis and initiates a severe hypermetabolic response (Stephens et al., 2016
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This approach helps relieve pressure within the abscess cavity and reduces the translocation of bacterial toxins into the bloodstream, thereby lowering the risk of SIRS and improving the patients overall clinical condition
Research-Ready Output for Biomarker & Mechanism Studies Data feed directly into metabolic pathway interpretation: fatty acid oxidation, branched-chain amino acid catabolism, mitochondrial dysfunction
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