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Here's why that combination is interesting: rapamycin inhibits mTOR, the cellular growth pathway that, when chronically overactivated, drives cellular senescence in follicle stem cell niches

Studies demonstrate rapid systemic uptake and targeted activity at endothelial and fibroblastic levels, implicating it in nitric oxide modulation, collagen synthesis, and vascular repair

This epigenetic modification subsequently drives PUFA biosynthesis, lipid peroxidation, and ferroptosis, as manifested by elevated LPO and Fe2 + levels alongside GPX4 downregulation (92)

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