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The retatrutide triple agonist GLP-1 GIP glucagon mechanism works through three parallel axes: GLP-1 receptor activation: Stimulates glucose-dependent insulin secretion from pancreatic beta cells, suppresses glucagon release from alpha cells, slows gastric emptying, and reduces appetite signalling in the hypothalamus

Next, stay for the diabetes tech show-and-tell as Diana demonstrates the specs of the latest pumps and sensors

Evidence Summary There is currently insufficient evidence of consistent magnesium depletion directly caused by GLP-1 therapy