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Multiple AMPK activators inhibit l-carnitine uptake in C2C12 skeletal muscle myotubes
In particular, a growing body of in vivo evidence supports the concept that some cancers become functionally dependent on FSP1, creating a therapeutic vulnerability that can be exploited either as monotherapy in specific contexts or as combination therapy with ferroptosis-inducing treatments
GSH preserves mTORC1 signaling activity and prevents the ISR during cystine deprivation and this protective function is supported by its role as a cysteine storage molecule
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