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Each mechanism of dysfunction at the pre-synaptic, post-synaptic, and peri-synaptic level offers itself as a target for intervention to improve stabilization, junctional signaling, and synapse integrity [213], potentially even before symptomatic weakness in those with genetic susceptibilities
Giustarini, D., Milzani, A., Dalle-Donne, I., & Rossi, R
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281,282 Liver-specific KO of upstream Hippo kinases, including MST1/2, LATS1/2, and the adaptor proteins SAV1 and MOB1a/b, leads to decreased YAP phosphorylation and increased nuclear translocation
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