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This novel feature of TNBS to wear off GSH, coupled with its potential for ROS generation, demonstrates that TNBS promotes two independent mechanisms of injury, namely, GSH limitation and ROS accumulation, which in cooperation trigger a chain of processes that lead to cell damage
The rectal administration of glutathione may provide a more direct route for the medication to enter the systemic circulation, potentially leading to better bioavailability and efficacy
Not everybody responds to the same blood pressure medications
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