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Its defining modification is a trans -3-hexenoyl group attached to the N-terminal tyrosine, a chemical change designed to protect the peptide from rapid cleavage by dipeptidyl peptidase-4 (DPP-4), the enzyme that quickly inactivates native GHRH
Picard, M
(PubMed) Rodriguez-Diaz E, Pearce EN
Similarly, metabolic abnormalities in PAECs, such as mitochondrial dysfunction and increased ROS production, promote endothelial inflammation and macrophage polarization through the release of metabolites like mtDNA and lactate (Rao et al., 2025)
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