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Genetically predicted glucose-dependent Insulinotropic polypeptide (GIP) levels and Cardiovascular Disease Risk are driven by distinct causal variants in the GIPR Region
Computational and mutational analysis of human glutaredoxin (thioltransferase): probing the molecular basis of the low pKa of cysteine 22 and its role in catalysis
The affected pathways clustered into several categories relevant beyond cosmetic applications: Extracellular matrix remodeling: Upregulation of decorin, multiple collagen types (I, III, VII), and matrix metalloproteinase inhibitors (TIMP-1, TIMP-2)
Similar efficacy of DPP-4 inhibitors to either metformin or pioglitazone was also shown in formerly drug-nave patients who did not reach glycaemic goals with non-pharmacological interventions
