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The oral capsule form, specifically the stable salt version, is designed to survive the gut and is often studied for its effects on the gastrointestinal tract and systemic healing
TP53 also influences ferroptosis sensitivity through other mechanisms, such as downregulating VKORC1L1 (vitamin K epoxide reductase complex subunit 1 like 1) in vitamin K metabolism [151] and inducing SAT1 (spermidine/spermine N1-acetyltransferase 1), an enzyme in polyamine catabolism that produces ROS [152]
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