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The anti-inflammatory mechanisms of GLP-1RAs include that they can reduce inflammation through inhibition of nuclear factor kappa B (NF-B) signalling, can reduce proinflammatory cytokine signalling (these cytokines are also released by mast cells), and can modulate immune cell activity
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Ten years later, in 1996, various peptide fragments of GIP were studied to investigate the potential for identifying a GIP receptor-specific antagonist
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