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J Genet Genom

Instead, they act as ATP-independent holdases that bind misfolded or partially unfolded proteins, preventing their aberrant aggregation and facilitating subsequent degradation

Knockdown of intracellular GPX4 can increase the level of intracellular lipid peroxides accompanied by ferroptosis, while iron chelator DFO and vitamin E prevent ferroptosis in GPX4 knockdown cells, reflecting the importance of GPX4 in protecting cells from excessive toxic lipid peroxides (Yang et al., 2014)

Regulatory functions of L-Carnitine, acetyl, and Propionyl L-Carnitine in a PCOS mouse model: focus on antioxidant/antiglycative molecular pathways in the ovarian microenvironment