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Experimental vascular studies indicate that BPC-157 modulates endothelial nitric oxide synthase activity by altering the interaction between endothelial nitric oxide synthase and its regulatory binding partner caveolin-1 [1][2]

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However, research also shows Bpc-157 suppresses Ki-67 and downregulates VEGF in some tumor models, suggesting the relationship is more complex than simple promotion of all growth