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Similarly, Iron-coordinated polymer nanoparticles (PCFD) further integrate doxorubicin (DOX) and cinnamaldehyde (CA) to disrupt redox homeostasis through triple mechanisms: Fe + -mediated GSH depletion, DOX-induced ROS generation, and CA-enhanced oxidative stress (202)
It can also upregulate Bax and downregulate Bcl-2 to inhibit the progression of the cell cycle from the G1 phase to the S phase, causing tumor cells to stay in the G1 phase and increasing DNA vulnerability to damage (166)
Increased D2 autoreceptor activity is associated with lower extracellular glutamate levels and reduced glutamate release in regions like the nucleus accumbens
As federal research funding remains uncertain, DMPI scientists say sponsored research agreements make up some of the gaps
