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These comparisons mix a single agent versus a combination, which makes riskbenefit decisions more complex and should be handled by a clinician
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Specifically, AMPK can inactivate ACC by phosphorylation, resulting in reduced activity of malonate monoacyl coenzyme A (CoA) and reducing allosteric inhibition of CPT1, thereby enhancing the activity of CPT1, which transports long-chain fatty acids from carnitine to mitochondria for -oxidation [17]
It clarifies its role
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