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The aim of this review is to analyze the literature on the impact of GSH deficiency on the etiopathology of schizophrenia, with particular emphasis on neurodevelopmental animal models in which oxidative stress during early postnatal development causes schizophrenia-like symptoms in adulthood
39), which revealed that the tumor tissues of mice treated with NP4 exhibited the strongest green fluorescence (apoptotic cells), demonstrating the good anti-tumor activity of NP4
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