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In parallel, sHSPs, ranging from approximately 12 to 43 kDa, have emerged as critical modulators of protein aggregation and toxicity in the nervous system (Fig

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Another potential mechanism for age-dependent loss of antioxidant genes was provided by one of the studies showing that the presence of transcription repressor BACH1 along with the absence of coactivator CREB-binding protein (CBP) in the aged mice may convert NRF2 binding from an active ARE to an alternative ARE [114]

This gradual dose escalation is designed to minimize common adverse effects, particularly gastrointestinal symptoms such as nausea, vomiting, diarrhea, and constipation