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IGF-I is harmonically produced by peaks that coincide with periods of neuron progenitor proliferation and differentiation, neuritic outgrowth (increasing the number of dendrites, axonal cones, synapse number) or post-injury conditions[59]

These results were consistent with previous findings and highlighted the significance of N-myristylation of FSP1, which could target to the plasma membrane, induce NADH-dependent reduction of CoQ, and finally suppresses lipid peroxidation, the hallmark of ferroptosis