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The gastrointestinal side effects of Zepbound stem from its mechanism as a GIP/GLP-1 receptor agonist, which slows gastric emptying and alters bowel motility
The glucagon component draws the most research interest, since GCGR activation is associated in laboratory models with hepatic glucose output, thermogenesis, and changes in hepatic fat fraction relevant to NAFLD and MASH studies
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Differences in formulation and dosing add uncertainty when combined with prescription therapy
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