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In pancreatic beta cells, semaglutide stimulates glucose-dependent insulin secretion, meaning it promotes insulin release only when blood glucose levels are elevated

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Multiple rodent models of PCOS, capitalizing mainly on the impact of androgen excess at different developmental windows, with variable penetrance in terms of reproductive and metabolic impairment, have been used in translational studies, in order to capture the phenotypic heterogeneity of the syndrome 38