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However, the direct vascular protective effects of GLP-1 RAs, including anti-inflammatory and endothelial actions, could theoretically operate independently of baseline metabolic status [31]
The median Cmax in this study is significantly lower than that considered therapeutic for preventing organ transplant rejection in humans, where a blood concentration of 8-15 ng/ml is often desired.[14, 53] This is intentional, as our goal was to use a dose that results in measurable blood concentrations, but not with the objective of causing immunosuppression
On weight the table says the GLP-1 effect dominates, with the DPP-4 inhibitor contributing only modest additional loss on top of it
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