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Posttranslational modification of cysteine in redox signaling and oxidative stress: focus on s-glutathionylation

Hyperoside (0.10.5% in HPL) directly inhibits adriamycin-induced mitochondrial dysfunction and podocyte injury through the inhibition of chondriokinesis in vivo and in vitro , thereby ameliorating renal and podocyte injury to reduce albuminuria in individuals with early diabetic nephropathy (Zhang et al., 2013

doi: 10.3389/fnut.2019.00093 74 ShiptonMJThachilJ

The ratio describes the relative distribution of GSH and GSSG, whereas redox potential also depends on their absolute concentrations, pH, and temperature