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Even physiological concentrations of soluble UA have been proved to induce gene expression of macrophage-secreted inflammatory cytokines and stimulate production of reactive oxygen species in mature adipocytes
In vivo and in vitro studies carried out in the brain of rats have demonstrated the role of NMDAR in the pathophysiology of HL deficiency
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For instance, while agents such as L-carnosine, omega-3 fatty acids, methyl-B12, and coenzyme Q10 demonstrated inconsistent or limited improvements in specific symptom domains (e.g., sleep disturbances, social cognition, gastrointestinal symptoms), others such as NAC and SFN revealed more consistent clinical benefits
