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Hepatic insulin resistance diminishes the ability of insulin to suppress gluconeogenesis and promotes de novo lipogenesis, driving lipid accumulation and pro-inflammatory signaling in hepatocytes ( Hepatic inflammation and fibrosis further aggravate systemic insulin resistance through the release of hepatokines such as fetuin-A and FGF21, which impair insulin signaling in peripheral tissues ( Given this mechanistic interdependence, the liver-pancreas axis represents an important therapeutic target

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Semaglutide, through its innovative mechanism of action that mimics the natural incretin hormone GLP-1, offers not just improved glycemic control but also significant weight loss benefits, a combination that has been elusive in diabetes management for decades