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Thus, the spectrum to consider is expansive, including Glu from GLP-1, Asp similar to Glu from GIP, Ala, and even Leu/Ile/Val (forming a hydrophobic area along with Phe 22 )
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Given the importance of NLRP3 inflammasome in the pathogenesis of fibrotic disease, the antifibrotic effects of GLP-1R could partially be attributed to its inhibitory potential against NLRP3 inflammasome activation
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