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Treatment with A 40 oligomers significantly reduced the cell viability and apoptosis of astrocyte brain cultures and increased oxidative modifications of DNA, lipids, and protein, enhanced pro-inflammatory cytokine release and increased the activity of the proteolytic matrix metalloproteinase enzyme, matric metalloproteinase (MMP)-2 and reduced the activity of MMP-9 after 24 h
ATP content was calculated as nmol/mg of protein
18 In kidney injury, extracellular vesicles and secreted lipid mediators are sufficient to synchronize ferroptotic death across spatially contiguous tubular epithelial cells, thereby contributing to a self-amplifying wave of tubular death during the transition from acute kidney injury to chronic kidney disease
di Ilio C et al
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