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Neuroinflammation and oxidative stress contribute to secondary brain injury

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We recruited 120 patients in the last 3 months of 2021 who had at least two symptoms consistent with Long COVID and had previously been diagnosed and treated for acute COVID-19 infection

Both Uncariae Ramulus Cum Uncis extracts and rhynchophylline inhibited KA-induced IL-1 and brain-derived neurotrophic factor (BDNF) gene expression, which were related to enhanced neuronal excitability after KA injection [40, 42, 43]