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These results provide a structural framework for understanding class B GPCR activation through hormone binding

With more receptors, neurons attempt to restore the signals to normal

In line with our short-term metabolic analyses, previous studies in New Zealand obese and diet-induced obese male mice suggested that GLP1/E acts primarily at central levels to reverse metabolic alterations by decreasing food intake and body weight, while the potential beneficial effects on pancreatic beta-cells were possibly secondary and indirectly-mediated 33,48

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