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Whilst the primary therapeutic targets are appetite-regulating centres, the widespread distribution of GLP-1 receptors means that semaglutide could theoretically influence neurological functions beyond appetite control

Endocr Rev 20:876913 Kieffer TJ, McIntosh CH, Pederson RA (1995) Degradation of glucose-dependent insulinotropic polypeptide and truncated glucagon-like peptide 1 in vitro and in vivo by dipeptidyl peptidase IV

Enhances protection against DNA damage, increases skin elasticity, function and detoxification of liver cell structures and contributes to the process of detoxification of the body from heavy metals and toxins

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