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First, there is no gold standard for detecting ferroptosis, especially in the small field of EBV reactivation, where the study of ferroptosis is still in its infancy

Given that the elevated striatal dopamine synthesis relates to better treatment response [11] in psychosis, the observed glutamatergic excess has been considered to be an index of reduced treatment responsiveness in psychosis [12]

In contrast, FAF2 (Fas-associated factor family member 2) interacts with ubiquitinated G3BP under HS treatment and is essential for SG disassembly following HS but not SA treatment ( 4 )

However, the body's ability to produce glutathione is not infinite