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GLP-1R activation inhibits NF-kB through a series of complicated actions inside the cell, but ultimately for our purposes this leads to the downregulation of multiple inflammatory cytokines, including but not limited to Tumor Necrosis Factor Alpha, (TNF-a) Interleukin 6 and 1-beta (IL6 and IL-1b) and multiple others

These findings provide direct evidence that S-Acetyl-L-glutathione activates programmed cell death in lymphoma cells, at least partially through the depletion of intracellular GSH

Accordingly, much of the heterogeneity in the existing literature may reflect differences in endpoint level, ascertainment strategy, and control of confounding, rather than the absence of a biological effect

However, patients should be monitored closely, especially when starting treatment or changing doses