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shear stress, a tangential force exerted parallel to the surfaces of cardiomyocytes, primarily arising from the frictional force of blood flow against the vessel wall[20]

Conversely, conditions of pressure overload, such as HFpEF, arterial hypertension, or aortic stenosis, typically induce concentric hypertrophy through parallel sarcomere replication and transverse myocyte growth, culminating in ventricular wall thickening[44, 45]

As a result, users can experience a quicker return to fitness activities, optimizing their training schedules

The type II form represents about 5% of all cases of CGD and is the result of mutations in the NFC2 gene which encodes the p67-PHOX protein