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4ac), revealing the essential role of CNS GIPR for the maximal weight loss response to combined GIPR blockade/GLP-1R activation
The expression and distribution of FFARs such as GPR40, GPR119, and GPR120 in L and cells and glucose, along with their dual effects on insulin and GLP-1 secretion, have made them attractive therapeutic targets, leading to the development of agonists for these receptors for the treatment of T2DM [150,151,152,153,154]
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