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CNS tissue has an exceptional ability to take up glutamate via excitatory amino acid transporters (EAATs), leading to a high level of substrate readily available to be converted to -KG (and subsequently 2-HG, with its potential for downstream tumorigenic effects.) This may explain why IDH1 mutations are highly prevalent in many CNS malignancies like GBM and could play an important role in tumor progression

Patients Who Stalled on Dual Agonists: Individuals who reached a plateau on a dual agonist despite optimal dosing may benefit from the added energy-expenditure and appetite-modulating effects of the third pathway

Effects of L-carnitine on serum triglyceride and cytokine levels in rat models of cachexia and septic shock

but banned for use in competitive sport and not authorized for compounding into human-use products